Orig Post jeffnixon.sportsblog.com | Re-Post Duerson Foundation 4/7/2016
C.T.E. (Chronic Traumatic Encephalopothy) Three big words and one small acronym for what has been labeled the “industrial disease” of NFL football players.
Ever since Dr. Bennet Omalu performed an autopsy of Mike Webster’s brain, and saw the build-up of an abnormal protein called tau, there has been a concerted effort by scientists and researchers to see if they can detect the disease in living persons and discover how much risk an athlete has of developing C.T.E. But there is also the big question of why some players seem far more susceptible to it than others. No one knows the answer to that question, yet. Dr. Bennet Omalu has gone on the record in the publication Frontiers in Neurology saying “diagnosis of CTE remains autopsy based,” there is a “lack of specific diagnostic criteria required for pre-mortem clinical diagnosis,” and there is “currently no accepted method of diagnosing CTE until post-mortem pathological analysis has been conducted.”
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Despite those statements and the relatively small body of science on C.T.E., 94 players have appealed the NFL Concussion Settlement because, for the most part, they want all of the symptoms that are associated with the disease to be covered – and therefore entitling players to monetary awards.
According to the Boston University Center for the Study of C.T.E., “the symptoms include memory loss, confusion, impaired judgment, impulse control problems, aggression, depression, anxiety, suicidality, parkinsonism, and, eventually, progressive dementia. These symptoms often begin years or even decades after the last brain trauma or end of active athletic involvement.”